-[TODO—
-[The sexual orientation node increases femininity and decreases AGP, so those pathways are anti-correlated; however, the fact that straight AGP men also vary somewhat in their degree of femininity; some informal accounts (link Sailer) have emphasized how masculine (even hypermasculine) AGPs are, but this seems wrong]
-[briefly mention ETLE]
-[Berkson's paradox is also a thing]
-[People who don't quite seem to fit the coarse taxonomy might still be explained by the graph and a threshold model]
-]
+Then the value of the sexual-orientation node is pushing the values of its children in _opposite_ directions: gay males are more feminine and less autogynephilic, and straight males are less feminine and more autogynephilic, leading to two broadly different etiological trajectories by which transition might seem like a good idea to someone, even while it's not that the two types have nothing in common. For example, this model predicts that among autogynephilic males, those who transition are going to be selected for higher levels of femininity compared to those who don't transition—and in that aspect, their stories are going to have _something_ in common with their androphilic sisters, even if the latter are broadly _more_ feminine.
+
+(Of course, it's also the case that the component factors in a liability-threshold model would negatively correlate among the population past a threshold, due to the effect of conditioning on a collider, as in the famous Berkson's paradox. But I'm claiming that the degree of bimodality induced by the effects of sexual orientation is substantially greater than that accounted for by the conditioning-on-a-collider effect.)